November 2018 — High-salt Recovered Sequences are associated with the active chromosomal compartment and with large ribonucleoprotein complexes including nuclear bodies. Genome Research vol. 28, n° 11, dir. {Cold Spring Harbor Laboratory Press} p. 1733-1746
,
December 2017 — HuR-Dependent Editing of a New Mineralocorticoid Receptor Splice Variant Reveals an Osmoregulatory Loop for Sodium Homeostasis. Scientific Reports vol. 7, n° 1, dir. {Nature Publishing Group} p. 4835
,
December 2017 — RNA-binding protein HuR enhances mineralocorticoid signaling in renal KC3AC1 cells under hypotonicity. Cellular and Molecular Life Sciences vol. 74, n° 24, dir. {Springer Verlag} p. 4587-4597
,
September 2014 — Hypertonicity Compromises Renal Mineralocorticoid Receptor Signaling through Tis11b-Mediated Post-Transcriptional Control. Journal of the American Society of Nephrology vol. 25, n° 10, dir. {American Society of Nephrology} p. 2213-2221
,
February 2019 — UCP1 transrepression in Brown Fat in vivo and mineralocorticoid receptor anti-thermogenic effects. Annales d’Endocrinologie vol. 80, n° 1, dir. {Elsevier Masson} p. 1-9
,
December 2016 — The cAMP pathway regulates mRNA decay through phosphorylation of the RNA-binding protein TIS11b/BRF1. Molecular Biology of the Cell vol. 27, n° 24, dir. {American Society for Cell Biology} p. 3841-3854
,
April 2010 — Gain of oncogenic function of p53 mutants regulates E-cadherin expression uncoupled from cell invasion in colon cancer cells.. Journal of Cell Science vol. 123, Pt 8, dir. {Company of Biologists} p. 1295-305
,
November 2012 — Cooperative Anti-Invasive Effect of Cdc42/Rac1 Activation and ROCK Inhibition in SW620 Colorectal Cancer Cells with Elevated Blebbing Activity. PLoS ONE vol. 7, n° 11, dir. {Public Library of Science} e48344
,
August 2009 — Variations in the colchicine-binding domain provide insight into the structural switch of tubulin. Proceedings of the National Academy of Sciences of the United States of
America vol. 106, n° 33, dir. {National Academy of Sciences} p. 13775-13779
,
April 2005 — Overexpression of Bcl-2 is associated with apoptotic resistance to the G-quadruplex ligand 12459 but is not sufficient to confer resistance to long-term senescence. Nucleic Acids Research vol. 33, n° 7, dir. {Oxford University Press} p. 2192-2203
,
February 2008 — Characterisation of P60 surface lipoprotein and its evaluation in a recombinant ELISA. Veterinary Microbiology vol. 128, 1-2, dir. {Elsevier} p. 81
,
June 2007 — Fluorescence-based melting assays for studying quadruplex ligands.. Methods vol. 42, n° 2, dir. {Elsevier} p. 183-95
,
2017 — Dependence of the Ce(III)/Ce(IV) ratio on intracellular localization in ceria nanoparticles internalized by human cells. Nanoscale vol. 9, n° 4, dir. {Royal Society of Chemistry} p. 1527-1538
,
June 2015 — Understanding the stability of DNA G-quadruplex units in long human telomeric strands. Biochimie vol. 113, , dir. {Elsevier} p. 125-133
,
April 2021 — PfGBP2 is a novel G-quadruplex binding protein in Plasmodium falciparum. Cellular Microbiology vol. 23, n° 4, dir. {Wiley} e13303
,
2018 — Binding properties of mono- and dimeric pyridine dicarboxamide ligands to human telomeric higher-order G-quadruplex structures. Chemical Communications vol. 54, n° 15, dir. {Royal Society of Chemistry} p. 1897-1900
,
July 2020 — Genome wide distribution of G-quadruplexes and their impact on gene expression in malaria parasites. PLoS Genetics vol. 16, n° 7, dir. {Public Library of Science} e1008917
,
December 2019 — Functional interplay between TFIIH and KAT2A regulates higher-order chromatin structure and class II gene expression. Nature Communications vol. 10, n° 1, dir. {Nature Publishing Group} p. 1288
,
April 2014 — Topology of a DNA G-quadruplex structure formed in the HIV-1 promoter: a potential target for anti-HIV drug development.. Journal of the American Chemical Society vol. 136, , dir. {American Chemical Society} p. 5249-52
,
2018 — CtIP fusion to Cas9 enhances transgene integration by homology-dependent repair. Nature Communications vol. 9, n° 1, dir. {Nature Publishing Group}
,